The optic tracts join the brachium of the superior colliculus, and then signals travel to the pretectal area of the midbrain. Thus, the right optic tract will contain temporal retinal fibers from the right eye, as well as nasal retinal fibers from the left eye. At the optic chiasm, nasal retinal fibers will cross to the contralateral side of the optic tract, and the temporal retinal fibers continue on the ipsilateral side. The optic nerve then forms the optic chiasm, which diverges into a left and right optic tract. These are the first steps of the pupillary light reflex afferent pathway. The optic nerve sends impulses to the brain for further processing and image recognition. These signals are then relayed to the bipolar cells, which interact with ganglion cells, which in turn coalesce to form the optic disc and optic nerve (CN II). Photoreceptor cells in the outer layers of the retina, which are called rods and cones, convert light stimuli into neuronal impulses.
Light travels through the cornea, anterior chamber, pupil, lens, and the posterior chamber, eventually reaching the retina. The pupillary light reflex constricts the pupil in response to light, and pupillary constriction is achieved through the innervation of the iris sphincter muscle. Explain the potential clinical impact that the early detection of an abnormal pupillary light reflex can have on a patient, and how the systematic approach could help evaluate and treat patients with abnormal pupillary reflexes to improve outcomes.Įyes allow for visualization of the world by receiving and processing light stimuli.Review the management considerations for patients with pathological pupillary light reflex abnormalities.Describe the different pathologies associated with abnormal pupillary light reflexes.Explain the mechanism of pupillary light reflex.This activity reviews the evaluation and management of the abnormal pupillary light reflex and highlights the role of the interprofessional team in caring for patients with abnormal pupillary reflexes. This is known as the pupillary light reflex.
Adjustments via the muscles connected to the lens, ciliary bodies, and muscles that make up the iris are stimulated by several nerves. This light interacts with the structures and nerves of the eye to create images. With the declining incidence of neurosyphilis, the sign is increasingly likely to indicate another cause, although an assiduous search for lues should also be undertaken.Eyes allow visualization of the world by receiving and processing the energy of light as it enters the eye. Magnetic resonance imaging studies have localized the lesion in patients with sarcoidosis and multiple sclerosis. Originally associated with tabes dorsalis, the sign has now been found in a number of conditions with lesions in the area of the nucleus of Edinger-Westphal. With the declining incidence of neurosyphilis, the sign is increasingly likely to indicate another cause, although an assiduous search for lues should also be undertaken.ĪB - The Argyll Robertson pupil, a miotic pupil that fails to react to direct light, has been described for more than a century.
N2 - The Argyll Robertson pupil, a miotic pupil that fails to react to direct light, has been described for more than a century. T1 - Significance of the Argyll Robertson pupil in clinical medicine
0 kommentar(er)
